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A myocardial infarction causes the death of massive numbers of heart cells. To repair the injury, the body replaces the damaged and dead cells with scar tissue. Initially, the scar tissue is ...
Microbubbles improve myocardial remodelling after infarction Date: February 21, 2013 Source: Universität Bonn Summary: Scientists have successfully tested a method in mice allowing the ...
The results show that the mice, following myocardial infarction, ... "There are attempts to treat the scar tissue with gene therapy or stem cells - by contrast, ...
Areas of thinned myocardium (≤5.5 mm) were previously thought to be regions of transmural infarction, without remaining viable tissue. However, new data using late-enhancement cardiovascular ...
A myocardial infarction causes the death of massive numbers of heart cells. To repair the injury, the body replaces the damaged and dead cells with scar tissue. Initially, the scar tissue is ...
The Universidad de Valladolid and the Centro Nacional de Investigaciones Cardiovasculares (CNIC) have developed a method for identifying the 3-dimensional features of scar tissue formed after a ...
In a new study, researchers from the University of Tsukuba have shown how cells in the scar tissue can be converted to heart muscle cells, effectively regenerating the injured heart.
A myocardial infarction, or heart attack, can be a complication of this, resulting from tissue damage or death that affects heart function. Both cause similar symptoms, though ischemia may be ...
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Drug combination shows promise for early treatment after myocardial infarction - MSNA myocardial infarction, commonly known as a heart attack, remains one of the leading causes of illness and death worldwide. ... It led to less collagen build-up in the scar tissue, ...
German scientists from the Bonn University Hospital successfully tested a method in mice allowing the morphological and functional sequelae of a myocardial infarction to be reduced.
A method was successfully tested by scientists from the Bonn University Hospital in mice which allowed the morphological and functional sequelae of a myocardial infarction to be reduced.
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